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Regulator of G-protein signaling 2 (RGS2) was downregulated in stressed female mice and women with major depressive disorder. Overexpression of the protein restored social approach in stressed mice.
A randomized controlled trial in patients with depression shows pharmacogenetic testing prior to treatment prescription could help some patients achieve better outcomes.
Large-scale changes in functional connectivity in the brain occur in patients with treatment-resistant depression administered targeted magnetic stimulation, which researchers believe could help predict treatment response.
A new mouse model shows links between depression, prolonged and severe stress, and an increase heart disease risk and response to lipid-lowering drugs.
Hereditary factors are partly responsible for childhood anxiety and depression according to the largest study of its kind in the world that looked at genetics from more than 65,000 children.
Alzheimer’s disease has long been linked to depression, but now a new study identifies common genetic factors in both conditions.
Through research done mostly in a mouse model, Canadian scientists have identified a biomarker, soluble E-selectin, that could be used to test whether women, but not men, have neurovascular signs of depression.
Researchers have developed a test that can not only indicate the presence of depression, but it can also indicate therapeutic response with a single biomarker called adenylyl cyclase.
The success of this proof-of-principle study rests on the researchers’ discovery of a specific pattern of brain activity that indicates the onset of symptoms (a neural biomarker) in the patient, and their customizing the deep brain stimulation device to trigger a response selectively when it detects the pattern that constitutes the biomarker.
A new approach developed at Emory University School of Medicine combines genome-wide association study data with human brain proteomic data and has identified 19 genes and 25 brain proteins that could cause depression.