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A low-protein diet disrupts a nutrient signaling pathway in cells that is a driver of colon cancer.
Discovery by scientists at the Institute for Research in Biomedicine, in Barcelona, reveals how the group of tumor cells responsible for relapse behaves and also the genes that define them.
Scientists have discovered that tumor stem cells with Mex3a protein activity remain in a state of latency that confers resistance to chemotherapy.
The researches showed in an animal model of colorectal cancer that using viral and bacterial vaccine approaches together is safe and far more effective at fighting the cancer than either approach by itself.
Previous research studies have demonstrated that circulating tumor DNA can be detected in blood and that the presence of ctDNA post-surgery predicts the risk of cancer recurrence.
These new findings could eventually inform the development of new therapeutics based on the small molecules produced by such beneficial bacteria.
The presence of high levels of a specific strain of Bacteroides fragilis in colon polyps increases inflammation and may encourage progression to colon cancer, suggests research from the University of Washington.
There is a clear link between taking antibiotics and an increased risk of developing colon cancer within the next five to ten years, according to Swedish scientists who believe the intestinal microbiome is thought to lie behind the increased risk of cancer.
City of Hope’s new findings are significant because immunotherapy has been seen as ineffective against microsatellite stable (MSS) colorectal cancer, which represents 95% of all metastatic colorectal cancer cases. These patients have few treatment options once they become resistant to chemotherapy.
The researchers examined the proposed link between peroxisome proliferator-activated receptor (PPAR) and how it promotes regeneration and tumorigenesis by enhancing intestinal stem cell numbers, proliferation, and function.